Type 2 Diabetes Variants in the SLC16A11 Coding Region Are Not Loss-of-Function Mutations (Cell Reports)

Zhao et al. elaborate on their recent paper by discussing how the physiological study of SLC16A11 function by using mouse models was designed and performed and its implications. This Matters Arising response paper addresses the Hoch et al. (2019) Matters Arising paper published concurrently in this issue of Cell Reports.